Gallic acid-related downregulation of epidermal growth factor (EGFR), phosphorylated serine/threonine kinase 1 (Akt/p-Akt), and phosphorylated extracellular signal-regulated kinase (Erk/p-Erk) signaling pathways has been proposed as a possible tool to induce suppression of cancer progression in vivo as well [41]. The gene discussed is AKT1; the disease is cancer.