These results, together with studies showing that the splicing activity of hnRNP H affects Ras-MAPK pathways in cancer [37] and the expression of translation factors [38], prompted us to investigate whether—and if so, how—hnRNP H/F affects mRNA translation by regulating the expression and/or the activity of translation factors, and to explore the consequences of these regulations in GBM. Here, HNRNPH2 is linked to glioblastoma.