These data suggest that, although DHT exposure from 3 weeks of age models the hyperandrogenism, anovulation and obesity of a PCOS‐like phenotype, it lacks the LH hypersecretion common to clinical PCOS, suggesting that exposure to androgen excess during different developmental windows can drive distinct PCOS‐like phenotypes. This evidence concerns the gene PLOD1 and obesity due to melanocortin 4 receptor deficiency.