Chen et al. [38] found that Sal might protect against myocardial fibrosis in mice with myocardial infarction (MI) by decreasing the expressions of TNF-α, TGF-β1, IL-1β, and Bax and increasing Bcl-2, VEGF, Akt, and eNOS. The gene discussed is TNF; the disease is myocardial infarction.