The interferon alpha beta signature, T helper pathway, and TCRA pathway of SSTR2-high groups were higher than those of SSTR2-low groups in most cancers (interferon alpha beta signature: 100%; T helper signature: 92.31%; and TCRA pathway: 100%, Figures 2I–K), suggesting that the SSTR2-high group has better T cell activation than the SSTR2-low group in most cancers. This evidence concerns the gene SSTR2 and cancer.