KLRK1 and X-linked immunodeficiency with magnesium defect, Epstein-Barr virus infection and neoplasia: Ravell et al showed a decrease in CD70, CD28, and TCR glycosylation in T cells of XMEN patients, perhaps indicating that in addition to the decrease in NKG2D, the loss of glycosylation of other signalling molecules may be important to the increase susceptibility to EBV in XMEN patients.