Lijingui et al.42reported that ARS reduced the excess production of pro‐inflammatory cytokines (IFN‐γ, IL‐17 and TNF‐α), increased the production of IL‐10, suppressed the level of serum aminotransferases and protected mice from Con A‐induced autoimmune hepatitis by inhibiting the phosphorylation of ERK, JNK and p38, NF‐κB p65 and IκBα signaling in a dose‐dependent manner. The gene discussed is NFKB1; the disease is autoimmune hepatitis.