These GLUTs are demonstrated to play an important role in cancer hyperglycolysis and sugar uptake leveraged by the Warburg effect.[26, 27, 28] Both results from GLUT1 transfected cell uptake and GLUT inhibitor mediated competitive accumulation assay support the mechanism of cellular transport and intracellular accumulation of Cor‐sugars is regulated at least by the tumor specific transporter GLUT1. The gene discussed is SLC2A1; the disease is neoplasm.