Other studies that focused on lncRNAs, such as myocardial infarction–associated transcript (MIAT) (203), myosin heavy-chain-associated RNA transcripts (Mhrt) (204), and H19/miR-675 (205), all similarly found that forced expression or overexpression of these lncRNAs lead to preservation of cardiomyocyte apoptosis involved in the pathogenesis of DCM. Here, H19 is linked to familial dilated cardiomyopathy.