The stipulated drug candidate used in this research, that is, OMT, was able to slow down the pathogenesis of glaucoma by not only lowering and further managing IOP but also substantially restoring the homeostasis of the retinal vasculature, which clearly identified the TGFβ1 isoform as a future target for therapies related to glaucoma inhibition (Figure 10). Here, TGFB1 is linked to glaucoma.