Impaired oxidative metabolism and a concurrent buildup of CD271 proteins in mdx mice observed in this study parallels the increased levels of nerve growth factor transcripts in patients of Duchenne Muscular Dystrophy, thus the authors suggested that hyperactivation of the CD271-associated pathway may rescue the slow-oxidative phenotype in the dystrophic pathology. Here, NGFR is linked to Duchenne muscular dystrophy.