Interestingly, a recent study discovered that treating GSD-1β patients with a sodium glucose cotransporter 2 (SGLT2) inhibitor, an anti-diabetic drug inhibits renal reabsorption of glucose while facilitating excretion of the inhibitory glucose analog, resolving the neutropenia and neutrophil dysfunctions (152). Here, SLC5A2 is linked to glycogen storage disease Ib.