ITGA2B and metabolic dysfunction-associated steatohepatitis: Considering the role of platelets receptors in NASH, it has been shown that the GPIIb subunit of the platelet fibrinogen receptor GPIIb/IIIa is not involved, confirming that platelets aggregation is not responsible for NASH (Malehmir et al., 2019), as well as platelet integrin α2β3 binding motif of fibrinogen (Kopec et al., 2017).