SNHG17 and gastric cancer: In their report [15], SNHG17 depletion inhibited cell proliferation, arrested the cell cycle in the G0/G1 phase, promoted apoptosis, and suppressed cell migration and invasion both in vitro and vivo. Mechanistically, SNHG17 acted as an oncogene in GC by epigenetically silencing p15 and p57 via activation of EZH2.