We observed that lack of PKCε in the p53-null tumour-prone model affected the age of tumour formation and survival, as Trp53-/-Prkce-/- mice developed lymphoblastic lymphoma earlier than the Trp53-/-Prkce+/+mice (17.6 weeks vs 19.6 weeks, on average). The gene discussed is PRKCE; the disease is lymphoblastic lymphoma.