For instance, in terms of LPS-triggered inflammation of acute respiratory distress syndrome (ARDS), miR-381-3p presents a low expression, and KCNQ1OT1 silencing inhibition may attenuate LPS-caused ARDS inflammation via up-regulating miR-381-30 and modulating ETS2[48]. Here, KCNQ1OT1 is linked to acute respiratory distress syndrome.