Moreover, the following parameters were identified as hospital-acquired AKI exposures: increases in inflammatory markers (CRP, Ferritin, Fibrinogen), glucose levels, markers of hypoxic damage and cell death (lactic acid and LDH), liver-pancreatic damage (bilirubin and amylases), presence of coagulopathy (APTT, INR, fibrinogen), and lower urinary pH. This evidence concerns the gene CRP and acute kidney injury.