To further evaluate the mechanism by which the ovarian exosomal miR‐122‐5p promotes apoptosis of ovarian GCs, the transcription levels of BCL9 and miR‐122‐5p in ovarian GCs, the level of miR‐122‐5p in ovarian GCs of the CTX group was significantly upregulated compared with that in the control group, while the expression of BCL9 was the opposite (Figure 6A). Here, BCL9 is linked to cerebrotendinous xanthomatosis.