VIM and rheumatoid arthritis: Notably, the biological role of the selected proteins corresponds to molecular mechanisms that make a significant contribution to RA pathogenesis, including the implementation of a nonspecific immune response and cell migration (α-enolase [35,36], protein-arginine deiminase type-4 [37], fibrinogen α-chain, fibrinogen β-chain [38]), reorganization of the actin cytoskeleton and cell filaments (C-X-Chemokine 10 [39,40], vimentin, actin), organization of chylomicrons, transport and/or metabolism of lipids (apolipoproteins A-I, C-III, and E, and serum albumin [41]).