Accumulating evidence suggests that dysregulated m6A modification is involved in the carcinogenesis and progression of multiple cancers; for example, dysregulated m6A modification in the transcripts of some oncogenes, such as Snail, or tumor suppressor genes, such as PHLPP2, is associated with tumor proliferation and metastasis (Liu et al., 2018a; Lin et al., 2019), and they have the potential for targeted therapies (Su et al., 2018). Here, SNAI1 is linked to neoplasm.