Now, we speculated that based on our results, the interaction of the lncRNAs PART1, EPAS1, and ACSL3 may promote EMT through PI3K-Akt signaling and TGF-β signaling pathways, thereby causing the occurrence of BCR in PCa, with ferroptosis as one of the important links in this process. This evidence concerns the gene PART1 and posterior cortical atrophy.