Now, we speculated that based on our results, the interaction of the lncRNAs PART1, EPAS1, and ACSL3 may promote EMT through PI3K-Akt signaling and TGF-β signaling pathways, thereby causing the occurrence of BCR in PCa, with ferroptosis as one of the important links in this process. The gene discussed is EPAS1; the disease is posterior cortical atrophy.