(2016), which has a higher half-life and showed high therapeutic activity against Stx2 toxicity in three different mouse models (single i.v. Stx2 lethal dose, several i.v. incremental Stx2 doses, and intragastric STEC infection), suggesting being a promising option to treating STEC infections to prevent or ameliorate HUS outcome (Mejías et al., 2016). The gene discussed is STX2; the disease is hemolytic-uremic syndrome.