Delta-tocotrienol (δT) treatment could similarly suppress the activity of SLC1A5 by derivatizing glutamate and glutathione, as well as some essential amino acids; while knockdown of circ-LDLRAD3 reduced the expression of SLC1A5 by sponging miR-137, which was eventually also observed to inhibit the development of NSCLC (23, 24). This evidence concerns the gene SLC1A5 and non-small cell lung carcinoma.