Sepsis-induced ARDS patients displayed FASLG−FAS, which reportedly is related to increased protein permeability in the pulmonary alveoli [27], and CCL3/4/5−CCR1, which is related to the chemokines and proinflammatory cytokines that participate in and promote inflammatory responses related to macrophages [28, 29]. This evidence concerns the gene CCL3 and acute respiratory distress syndrome.