Sepsis-only patients displayed IL1B−IL1RAP, of which Interleukin-1-B (IL1B) is a proinflammatory cytokine that plays an important role in sepsis and affects the p38 MAPK and NFκB signaling pathways [30, 31], and TGFB1−TGFBR1, of which transforming growth factor beta1 (TGF-beta1) gene single-nucleotide polymorphisms and plasma TGF-beta1 levels were thought to be associated with susceptibility to sepsis [32]. The gene discussed is NFKB1; the disease is Sepsis.