As shown in Figures 2(a) and 2(b), KEGG enrichment indicated that the DE-NRGs were mainly involved in the following pathways: (1) JAK-STAT signaling pathway; (2) diseases involving necroptosis, such as influenza A and systemic lupus erythematosus; and the (3) NOD-like receptor signaling pathway. The gene discussed is SOAT1; the disease is systemic lupus erythematosus.