In non-small cell lung cancer cells (NSCLC), the UA has been found to inhibit autophagy and suppress TGF-β1-guided EMT caused by the regulation of integrin αVβ5/MMP signaling, which is considered to be the reason for its anticancer activity in NSCLC cells [14–16]. Here, TGFB1 is linked to non-small cell lung carcinoma.