Notably, among females, there was a significantly decreasing trend in the percentage of MAFLD, liver steatosis, and fibrosis in the NP group (P = 0.004, P < 0.001, P = 0.045, respectively) but increasing trend in the HP group (P = 0.004, P = 0.019, P = 0.002, respectively) across serum PRL quartiles (Figure 3B), whereas such significant association was not shown among males (Figure 3A). The gene discussed is PRL; the disease is fatty liver disease.