It has been revealed that ginsenoside Rk1 and Rg5 suppressed the EMT process of lung cancer induced by TGF-β1 [22], and ginsenoside Rb2 could enhance E-cadherin, but weaken expressions of N-cadherin, vimentin and snail by mediating the TGF-β1/Smad signaling pathway [23]. Here, TGFB1 is linked to lung cancer.