Nevertheless, it was found that the admission level of CgA, which is a neurohumoral marker, was lower in the TTS group compared to patients with anterior ST-elevation myocardial infarction (STEMI), and the CgA level in TTS patients was positively correlated with NT-Pro-BNP, indicating that CgA is co-secreted with the B natriuretic peptide (BNP), and CgA may be a local factor expressed by cardiomyocytes, which interact with intracardiac catecholamines [65,66,67,68]. Here, CGA is linked to ST Elevation Myocardial Infarction.