ENDOG and infection: The absence of alteration in the gene expression levels of TRAIL and TNF receptor-associated factor 1 and 2 genes in combination with the significant up-regulation of Endonuclease G (EndoG) at 3 and 24 hpi in SSN-1 cells indicate that the RGNNV infection potentially induces apoptosis of SSN-1 cells via the mitochondrial pathway instead of following the extrinsic pathway [36].