Overall, our results revealed only a modest impact of IL-10 or TGF-β on moMΦ responses to ASFV, with a small, but statistically significant, increased ability of NH/P68 ASFV to infect moMΦ pre-treated with IL-10 at 24 hpi (using an MOI of 0.01) and with TGF-β tuning down the release by moMΦ of pro-inflammatory chemokines in response to NH/P68 ASFV infection, although without statistical significance. The gene discussed is TGFB1; the disease is infection.