Involvement of monocytes/macrophages in both the inflammatory and regenerative phases of post-infarction myocardial remodeling has already been confirmed by the presence of their prolonged CD68+, CD163+, CD206+ and stabilin-1+ macrophage infiltration in both IA and non-IA of the myocardium in patients who experienced a fatal MI [10]. The gene discussed is CD163; the disease is myocardial infarction.