The observed cytokine storm in SARS-CoV-2 infection, the uncontrolled release of proinflammatory mediators, such as TNF, IL-6, TNFα, IL-1β, IL-8, CCL2, CCL3, chemokine ligand CXCL10 and α-chain of IL-2 receptor, might be clinically associated with ARDS, diffuse alveolar damage, hypercoagulation and increased levels of acute phase reactants [28,29,30]. This evidence concerns the gene CCL2 and acute respiratory distress syndrome.