However, in the presence of antimicrobial peptides (AMP), such as LL37 (or CAMP), B-defensins and Ribonuclease 7, inert self-DNA can be converted into potent autoantigen triggering IFNα production by pDCs, and driving autoimmunity in psoriasis [12,36,37,38]. The gene discussed is CAMP; the disease is Autoimmunity.