These differences also translated into changes in tumour immune cell infiltrates, with the non-antibiotic treated LTS FMT group showing a CD8+ T-cell rich environment, whilst the STS and healthy control FMT groups demonstrated increased numbers of CD4+FOXP3+ T-regs and myeloid derived suppressor cells, both of which are associated with dampening of immune responses, thereby permitting tumour growth [16]. The gene discussed is CD4; the disease is neoplasm.