The tumour-localised expression of CXCL11 by an oncolytic vaccinia virus in a mouse model of mesothelioma successfully increased the trafficking of endogenous cytotoxic T lymphocytes to the tumour and induced systemic anti-tumour immunity, highlighting the importance of the CXCR3 axis for lymphocyte migration [48]. This evidence concerns the gene CXCL11 and neoplasm.