The tumour-localised expression of CXCL11 by an oncolytic vaccinia virus in a mouse model of mesothelioma successfully increased the trafficking of endogenous cytotoxic T lymphocytes to the tumour and induced systemic anti-tumour immunity, highlighting the importance of the CXCR3 axis for lymphocyte migration [48]. Here, CXCR3 is linked to neoplasm.