Despite these carcinogenic properties, when the p53 protein has not mutated, it acts as a tumor-suppressing protein; it is a nuclear phosphoprotein that has an impact on the regulation of a wide range of reactions, such as genotoxic cellular stresses, leading to multiple responses including the stimulation of DNA repair, initiation of cell cycle arrest and apoptosis [30]. This evidence concerns the gene TP53 and neoplasm.