KEGG analysis revealed that GSEA positive was mainly related to immunity, inflammation, and cell phagocytosis (for example, autoimmune thyroid disease, graft versus host disease, allograft rejection, internal immune network for IgA production, antigen processing and presentation, B cell receiver signaling pathway, Fcγ receptor-mediated phagocytosis, and natural killer cell-mediated cytotoxicity), and GSEA negative was mainly related to oxidative phosphorylation and neurodegenerative diseases (such as Parkinson’s disease and Huntington’s disease) (Figure 4A). This evidence concerns the gene CD79A and juvenile Huntington disease.