TRPV1 and endometriosis: The Wang group [33] also used fat as a control tissue in their rat model of endometriosis and showed that expression of the TRPV1 channel was increased in the dorsal root ganglia adjacent to endometrial tissue lesions but not those formed from fat, providing a plausible mechanism by which pain pathways are altered by endometriosis lesions that would be in agreement with our results.