CD68 and triple-A syndrome: A reduction in infiltrating CD68 positive cells in the aneurysm wall has previously been observed after CAG treatment in aneurysms in mice induced by local elastase wrapping around the abdominal aorta, and in mice where the component 3,4-benzopyrene (an active ingredient in cigarettes) enhanced angiotensin II AAA two weeks, but also six weeks, after AAA induction [19,42].