Elevated levels of inflammatory cytokines (e.g., TNFα, CXCL10), and inflammatory markers of tissue damage (such as C-reactive protein (CRP), lactate dehydrogenase, aspartate aminotransferase, gamma-glutamyl transferase, and N-terminal-pro-brain natriuretic peptide) were negatively correlated with gut dysbiosis in patients with severe COVID-19 [9]; this suggests that altered microbiota communities could play roles in regulating immune responses and disease severity in COVID-19. The gene discussed is CXCL10; the disease is COVID-19.