Early acquisition of somatic mutations in GATA1, resulting in the production of N-terminally truncated short form of GATA1 protein (GATA1s) [5,6,7], is linked to the induction of transient abnormal myelopoiesis (TAM) seen in 1 of 10 DS infants [8,9]. This evidence concerns the gene GATA1 and Dravet syndrome.