The effects observed of TGFβ in glucose uptake disagree with previously published results in which it had been described that TGFβ induces GLUT1 expression, glucose uptake and glycolysis [57]; these discrepancies could be explained by the type of GBM cells used in the study, as GSCs cells rely mainly on oxidative phosphorylation while differentiated glioma cells have a glycolytic phenotype [62]. This evidence concerns the gene SLC2A1 and glioblastoma.