STT3B and glioblastoma: Specifically, dolichyl-diphosphooligosaccharide–protein glycosyltransferase subunit STT3A and dolichyl-diphosphooligosaccharide–protein glycosyltransferase subunit STT3B were about 120- and 43-fold increased for GBM versus controls, indicating higher glycosylation in GBM.