A total of 45 genes were commonly dysregulated following both exposure durations, with two pathways implicated across all exposure concentrations and all exposure durations: cancer and organismal injury and abnormalities, both of which included genes such as tyrosinase-related protein 1b (tyrp1b), sequestosome 1 (sqstm1), pyruvate dehydrogenase kinase 2 (pdk2), and heterogeneous nuclear ribonucleoprotein L (hnrnpl). Here, PDK2 is linked to cancer.