FAK exerts its functions through the phosphorylation on the corresponding downstream target proteins in the cytoplasm, thereby enhancing tumor cell adhesion and promoting tumor growth, cancer-stemness, invasion, and metastasis ability [6, 7], promoting cancer cell epithelial to mesenchymal transition (EMT), tumor angiogenesis, chemotherapeutic resistance, and fibrosis in the stroma. This evidence concerns the gene PTK2 and neoplasm.