KLF4 and Hepatic fibrosis: A subsequent study reported that miR‐103‐3p, which is present at increased levels in exosomes from LPS‐treated THP‐1 macrophages, promotes the proliferation and activation of hepatic stellate cells (HSCs) by targeting the 3’UTR of Krüppel-like factor 4 (KLF4), thus exacerbating the progression of liver fibrosis (13).