In a previous study, which isolated mixtures from GC patient tumour tissue samples with at least 5% of NKG2A+ (KLRC1) tumour‐infiltrating T lymphocytes (TILs), HLA‐E‐KLRC1 interaction negatively affected IL2 receptor–dependent CD8+ TIL proliferation and contributed to CD8+ TIL dysfunciton.111. The gene discussed is HLA-E; the disease is neoplasm.