Our finding that the interaction between FET‐FOPs and BRD4 is mediated by the SWI/SNF complex, suggests that the interactions between other fusion oncoproteins and BRD4, and at least part of the oncogenic effect, could be mediated via the SWI/SNF chromatin remodelling complex also in those tumour types. The gene discussed is SMARCA1; the disease is neoplasm.