Previous studies have found several molecules that can anti‐inflammatory and /or neuroprotective in the setting of AD, including 5‐lipoxygenase, CatB, and receptors including class A scavenger receptors and toll‐like receptors (Heneka et al., 2015; Wu et al., 2017; Yan et al., 2021), almost all of which have normal physiological functions and whose inhibition may cause side effects. The gene discussed is TYRP1; the disease is Alzheimer disease.